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Activated AMPK shifts cells into an energy-efficient mode enhancing glucose uptake, fatty-acid oxidation, and mitochondrial respiration while downregulating fat storage and gluconeogenesis [2]

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For instance, LAS17, one of the most effective known inhibitors, has an IC 50 of 500 nM and works by covalently binding to tyrosine residues on GSTP1 (Louie et al

The transdermal composition of claim 2 which provides at least one of the following release pattern in an In Vitro study employing dialysis membrane and a phosphate buffer of pH 7.4 i) at least 30 % release in 24 hrs and 70 % release in 72 hrs