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465 However, lipid overload and prolonged lipid accumulation can induce hepatocyte apoptosis and increase endoplasmic reticulum (ER) stress

Mitochondrial metabolism of reactive oxygen species

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The peptide functions as a precision senolytic agent by competing with endogenous FOXO4 for p53 binding, releasing the tumor suppressor protein to trigger apoptosis specifically in cells that have accumulated the senescence-associated secretory phenotype (SASP) markers

F.Jr.et al (2001)

Moreover, consuming a HFD increased protein S -glutathionylation (protein-SSG formation) in the liver